Tag Archives: antioxidants

I Love Pomegranate Juice

9 May

PomegranateI love the pomegranate tree. I love pomegranate Juice. Every year we wait patiently for the large red fruits to ripen. What a fantastic taste delight.

We have been tending our pom trees for years and spend the early winter months drinking the pomegranate juice and making pomegranate ice cream.

The pomegranate, my favorite fruit, has recently made research news, and I suspect folks will become more aware in its remarkable health giving benefits.

I Love My Arteries: Carotid Care
View Animation: Cardiovascular Health

I HAVE READ THE Research DONE AT the Rambam Medical Center in Haifa, Israel WHO treated patients with carotid artery stenosis using FRESH pomegranate Juice for three years and the results were nothing short of remarkable.

The carotid arteries are the two large vessels that run along either side of your neck and supply the brain with blood.

Blockage if these arteries is called Stenosis, of these which is simply a narrowing of the blood flow due to a buildup of deposits along the arterial wall.

Carotid artery narrowing often goes unnoticed for years. Individuals with the problem may not experience any symptoms, though some start to complain that they hear a “swishing” noise with each heartbeat, which is due to blood swirling past the blockage.

A more common complaint is eventually this narrowing can lead to a mini stroke. This occurs when small pieces of the fatty deposits lining the arteries break loose and block small blood vessels further up in the brain. The result can be memory loss, temporary blindness, numbness, tingling, and muscle weakness.

Fortunately, the effects are generally temporary, but these events are often just a prelude to a bigger, full-blown serious stroke.

Normal treatment for carotid artery blockage falls into basically two categories. If the blockage is less than 70 percent, the treatment is to keep the blood thin and flowing with aspirin, anticoagulants, and other drugs-and hope nothing “breaks loose.” If The blockage is over 70 percent; surgery to clear out the blockage is often the course of action.
Many folk who have blockage of the artery also have narrowing elsewhere in the body, too. It’s a systemic problem, so there is likely some degree of blockage in the arteries of the heart and elsewhere.

Reading the research

Reading the research done on pomegranate juice is exciting and may be the perfect treatment for removing the blockages in the carotid arteries.

It seems that atherosclerosis is best addressed through diet and nutrition. And that’s where pomegranate juice has been shown to be a tremendous help.

Pomegranate Tees and GiftsGo figure! My favorite juice is not only delicious makes tasty ice cream it also helps my arteries stay healthy.

Everyday is an opportunity to nurture your body-mind, every day is an opportunity to tend your garden, and to nurture all that nurtures you in return.

Now that’s our grokking Numian way to live.
My secret of aging with wellness.

HAPPINESS IS: A daily jogging, grazing the greens from our gardens, and meals made from garden grown veggies.

Pomegranate Juice the Miracle Nectar

Dr. Michael Aviram, with the Lipid Research Laboratory at Rambam, thinks it’s possible that many high-risk cardiovascular patients can be spared both bypass and carotid surgery by simply drinking pomegranate juice.

Why Pomegranates Are Better Than Red Wine

NEW YORK, N.Y. and HAIFA, Israel, May 4, 2000 — In a strong confirmation of the power of pomegranates to fight heart disease, studies of healthy human subjects and mice with atherosclerosis showed why even moderate consumption of pomegranate juice could have significant clinical results.

According to studies at the Technion-Israel Institute of Technology, the cholesterol oxidation process -– which creates atherosclerotic lesions that narrow arteries and result in heart disease — was slowed by as much as 40 percent when healthy subjects drank 2-3 ounces of pomegranate juice a day for two weeks. Further, the juice reduced the retention of LDL, the “bad” cholesterol that after its oxidation aggregates and forms atherosclerotic lesions.

The study is published in the May issue of the American Journal of Clinical Nutrition.

“Pomegranates are proving to be the most powerful antioxidant available, better than red wine, tomatoes, vitamin E and a variety of other headline makers,” says Prof. Michael Aviram of the Lipid Research Laboratory at the Technion-Israel Institute of Technology, who led the team. Prof. Aviram, who was the first to prove the beneficial effects of red wine on cholesterol oxidation in humans, is an internationally recognized authority on the effect of food on heart disease.

Earlier, pomegranate juice was tested on mice from a special strain susceptible to atherosclerosis. When these mice were fed pomegranate juice for 11 weeks, their arteries had only half as many lesions as did the arteries of the control mice who got no juice. This strengthened the evidence that the juice would also slow or prevent the formation of lesions in humans.

Most recently, using sections of excised human arteries, Prof. Aviram showed that the active ingredient in pomegranates not only slows down cholesterol oxidation but actually minimizes the retention and aggregation of LDL cholesterol, the “bad” cholesterol, two additional processes that create atherosclerotic lesions. These lesions are minimized if the cholesterol is not retained, oxidized and subsequently aggregated.

When the subjects stopped drinking the pomegranate juice, the beneficial effects lasted for about a month.

Aviram chose pomegranates for his study because the fruit has long been in use in folk medicine in the Middle East, Iran and India for treating disease and infections, and he suspected that some of its medicinal value could be due to antioxidants.

Dr. Aviram’s recent study involved 19 patients (5 women and 14 men, 65-75 years old) with severe carotid artery stenosis.

Ten were selected to receive 250 mL (8.3 ounces) of 100 percent pomegranate juice daily, and the other 9 were given a placebo.

“Several tests were performed on the different groups. The primary one Utilized ultrasound to measure any changes in the thickness of the walls of the carotid arteries.

After one year, those not consuming the pomegranate juice had a 9 percent increase in thickness of their carotid arteries, meaning their arteries were becoming even less resilient. Those consuming the juice showed a decrease in thickness of 35 percent. These changes began to show up rather quickly. There was a 13 percent reduction in just the first three months of drinking the juice. And there were other benefits, as well. While the blood pressures of those on the placebo didn’t change, the systolic pressure (top number) of those on pomegranate juice went from an average of 174 to 162 mm Hg in just a month. And it reduced even further, to an average of 152 mm Hg at twelve months.

Other tests indicated that the juice drinkers had lower blood levels of oxidized cholesterol and more antioxidants. Even though this study was relatively small, the results are quite remarkable and confirm the similar findings of previous studies. In one earlier study, atherosclerotic lesions in mice were reduced in size by 44 percent by supplementing the diet with pomegranate juice.

The Promise of Pomegranate

An important branch of his research focuses on the protective role against oxidative stress and atherosclerosis development played by antioxidants and paraoxonases. Dietary antioxidants studied by Aviram include the polyphenolic flavonoids found in red wine, pomegranate, licorice, ginger and olive oil; as well as vitamins E and C, beta-carotene, and tomato’s lycopene. HDL (“the good cholesterol”) – associated paraoxonase was shown by Aviram to act as a second line of defense against oxidative stress, by hydrolyzing specific oxidized lipids.

Pomegranate juice has exhibited some of the strongest antioxidant activity of any food. For thousands of years, pomegranates have been used medicinally particularly in the Middle East. In fact, the healing powers of pomegranates have been mentioned in the Bible, Greek mythology, and ancient Chinese literature. Some scholars believe that the fruit referenced in the Garden of Eden story was actually a pomegranate. It may be new to our society, but researchers are just rediscovering one of the ancient health secrets known to our ancestors thousands of years ago.

Pomegranate juice may also prove to be promising in the treatment/prevention of breast and prostate cancer as well as in the treatment of diabetes. Additionally, one of the more unusual uses of pomegranate juice has to do with preventing the spread of AIDS.
Since the AIDS pandemic continues to spread researchers have started to look at topical microbicides in the form of creams or suppositories to block the entry of HIV into cells.
Efforts are underway to discover a microbicide that would be acceptable, accessible, affordable, and able to be moved quickly from the development stage to the millions of people who need it.
Researchers evaluated numerous juices and their ability to block HIV infections, pomegranate juice showed remarkable potential. It was not only safe, stable, and economical, it was also more effective than any other juice tested.

Pomegranate juice is rich in tannins, phenolic acids, polyphenols, and flavonoids. It is also known to contain a rare fatty acid, punicic acid that is structurally related to conjugated linolenic acid.

If you’re looking for paradise on Earth, Grow a pomegranate tree, plant fig plants, tend your organic gardens and go solar.

Now let’s relax, watch the sunset and have a glass of red wine or pomegranate juice together.
Tony Crow, Numian Institute

THE SCIENCE : This randomized controlled study is the FIRST! to show that pomegranate juice may reduce the amount of plaque in the arteries of patients with heavy plaque buildup (severe carotid artery stenosis) as well as substantially benefiting several important blood parameters.

Ten patients consumed 8 oz a day of POM Wonderful pomegranate juice for 1 year. Nine patients who did not consume pomegranate juice served as controls.

The thickness of the carotid artery wall (intima-media thickness, IMT) was measured and blood samples were taken at the beginning of the study and at 3, 6, 9 and 12 months. After 1 year, those patients who did not consume pomegranate juice showed a 9% increase in IMT, while those consuming juice showed a decrease of 30%. Furthermore, for those on pomegranate juice, systolic (but not diastolic) blood pressure was reduced by 21%, total antioxidant status of the blood increased by 130%, LDL oxidation decreased by 90%, antibodies to oxidized LDL decreased by 19% and serum paraoxonase 1 (PON1) increased by 83%. Major blood biochemical markers were not affected, including levels of LDL and HDL cholesterol.

Benefits were maintained in five patients who continued pomegranate juice treatment for 3 years, with further improvements in serum lipid peroxidation.

Pomegranate juice consumption for 3 years
by patients with carotid artery stenosis
reduces common carotid intima-media thickness,
blood pressure and LDL oxidation

Pomegranate Juice Research Report

Oxidative stress, a major contributor to cardiovascular diseases, is associated with lipid peroxidation in arterial macrophages and in lipoproteins.1–4 Oxidized low density lipoprotein (Ox-LDL) has been shown to be atherogenic and inhibition of LDL oxidation by potent dietary flavonoid antioxidants4,5 attenuated atherosclerosis development in laboratory animals.

Recently, it was shown that vitamin E supplementation to patients with carotid artery stenosis inhibited LDL accumulation in arterial macrophages.6 Protection of lipids from oxidation can be also achieved by serum paraoxonase 1 (PON1), an HDL-associated esterase that can hydrolyze and reduce specific lipid peroxides in arterial cells and in lipoproteins in coronary and carotid lesions.


The medicinal properties of pomegranate are described by all major religions and by folk medicine.11 Pomegranate juice (PJ) was indeed shown recently to possess impressive antioxidative properties due to its polyphenolics, tannins and anthocyanins.

We have recently shown the antioxidative and antiatherogenic characteristics of PJ consumption in atherosclerotic apolipoprotein E deficient (E0) mice.

Also, in healthy humans, PJ consumption was shown to possess potent antioxidative capabilities against lipoprotein oxidation, and increased serum PON1 activity and serum total antioxidant status.13 In the present study, thus, we analyzed for the first time the effects of PJ consumption by patients with carotid artery stenosis, on their serum oxidative stress in association with the progression of carotid atherosclerotic lesions.

Subjects and methods

Nineteen patients from the Vascular Surgery Clinic, 5 women and 14 men, aged 65–75 years, nonsmokers, with asymptomatic severe carotid artery stenosis (CAS, defined as 70–90% stenosis in the internal carotid arteries) were included in the study. These patients had an abnormal echo Doppler of the carotids, which was performed following a finding of carotid ‘‘bruit’’ on physical examination, or complains of headache or dizziness.

The patients were randomized to either pomegranate juice or placebo, and they signed an informed consent before the beginning of the study. Ten patients were included in the PJ treated group. Nine patients that did not consume PJ served as a control group.

Both groups were matched, with similar serum concentrations of lipids and glucose, and with similar blood pressures (data not shown). Both groups were treated with similar hypocholesterolemic and anti-hypertensive drugs. In each group, 60% of the patients were treated with statins, 60% were treated with angiotensin converting enzyme (ACE) inhibitors, 20% were treated with b-blockers, and 20% were treated with calcium channel blockers.

The patients continued their therapy along the study, and their dietary habits and life style did not change during the whole study. Ten patients consumed 50 ml of PJ per day (which contain 1.5 mmoles of total polyphenols) for a period of 1 year, and five out of them agreed to continue for up to 3 years. This PJ concentration was chosen based on our previous study on the beneficial PJ properties in healthy volunteers.13 Blood samples were collected after 12 h fast.

Blood analyses and echo doppler of the carotid arteries were performed at the beginning of the study and 3, 6, 9, 12, 22, 28 and 36 months after PJ consumption. In the control group echo doppler of the carotid arteries was performed at the beginning of the study and after 1 year.

The study was approved by the Helsinki Committee of the Rambam Medical Center, Israel Ministry of Health.

Pomegranate processing

Pomegranates were picked by hand, washed, and stored in tanks. The fruits were crushed, and squeezed. The juice was filtered, pasteurized, concentrated and stored at -18C

ach day along the study period, the concentrated PJ was diluted 1:5 (v:v) with water in order to obtain a single strength PJ.

The antioxidant composition of the juice include: 1979 mg/l of tannins (1561 mg/l of punicalagins and 417 mg/l of hydrolyzable tannins), 384 mg/l of anthocyanins (delphinidin 3,5-diglucoside, cyanidin 3,5-diglucoside, delphinidin-3-glucoside, cyanidin 3-glucoside, pelargonidine 3- glucoside), and 121 mg/l of ellagic acids derivatives. The juice contained also 3mg of vitamin C per 100 ml of PJ.

Atherosclerotic lesion analysis by B-mode ultrasonography

Common carotid artery IMT from B-mode ultrasound is a widely used measure of early atherosclerosis.

14–16 After careful axial scan, longitudinal B-mode images of common carotid artery wall boundaries were obtained with a high resolution color power Doppler ultrasound (ATL 5000 or 3500 Advanced Technological Laboratories, Bothell, WA) with a 5–12 MHz multifrequency transducer. IMT was electronically measured at the far wall of the distal common carotid arteries, about 1 cm from the carotid bifurcation, by assessing the boundaries of intima and media with electronic calipers.

Atherosclerotic plaques at the common carotid arteries and the carotid bulb, as well as the proximal and distal internal carotid arteries were imaged and the length and width of the plaque were assessed.

On duplex examination of the internal carotid arteries, flow velocities were calculated at the stenotic sites, and expressed by peak systolic velocity (PSV), and end diastolic velocity (EDV).

The ultrasound outcome analyses were the change over time in IMT, which was measured in the same preselected carotid artery segments, the change in the plaque dimensions and the change in blood flow velocities. A protocol was adapted in order to ensure that the arteries were examined from the same angle (601) at all followup examinations.

Patients had up to eight duplex examinations of the common and internal carotid arteries on each side: there was one ultrasound Doppler examination at baseline, and seven more examinations during PJ consumption.

All ultrasound studies were done by the same physician (DG), assuring reproducibility of the site of IMT and plaque measurement, as well as site and interrogation angles on the duplex follow-up examinations. In order to avoid potential introduction of scannerdependent variabilities, the same ultrasound system was used for individual patients on all followup examinations.

Analytical methods

All biochemical determinations were performed in serum. Blood glucose was measured using enzymatic kit (Roche). Total cholesterol, high density lipoprotein (HDL) cholesterol and triglyceride concentrations in serum were measured by diagnostic kits (Raichem).

Serum apolipoproteins A-I and B- 100 concentrations were determined using specific antibodies by an immunoturbidimetric assay.17 Serum paraoxonase 1 (PON1) arylesterase activity Serum arylesterase activity was measured using phenylacetate as the substrate. Initial rates of hydrolysis were determined spectrophotometrically at 270nm.

1.0 mmol/l phenylacetate, and 0.9 mmol/l CaCl2 in 20mmol/l Tris HCl, pH 8.0. Non-enzymatic hydrolysis of phenylacetate was subtracted from the total rate of hydrolysis. The E270 for the reaction is 1310M1 cm1. One unit of arylesterase activity is equal to 1 mmol of phenylacetate hydrolyzed/min/ml.18

Serum total antioxidant status (TAS)

Total antioxidant status was measured in serum with a commercially available kit (Randox Laboratories, Antrim, United Kingdom, catalog no. NX 2332).

Serum anti Ox-LDL antibodies

Serum anti Ox-LDL antibodies concentration was measured in samples collected from the patients before treatment and after 3 or 6 months of PJ consumption, by using the immunoelisa anti-Ox-LDL test (Immco Diagnostics, Inc. Buffalo, NY, USA, Cat No 1158). Results are expressed as Enzyme Units per milliliter (EU/ml).

Serum lipids peroxidation

Serum lipids peroxidation was measured before and after 12, 22, 28 and 36 months of PJ consumption. Plasma samples were incubated without or with 100mM of 2.2’-azobis, 2-amidinopropane hydrochloride (AAPH, Wako, Japan) for 2 h at 371C.19 At the end of the incubation period, the amount of lipid peroxides was measured by the method of Pomegranate juice consumption attenuates carotid artery stenosis 425 El-Saadani et al.20 Plasma lipids peroxidation was calculated by subtracting the values obtained in the absence of AAPH.

LDL isolation

Serum samples were drawn from the patients before and after 1, 3, 6, 9 and 12 months of PJ consumption and kept frozen at 701 until all the samples were collected.

Blood was collected also from healthy volunteers in the same time periods, for standardization of the assays. LDL was isolated from the frozen plasma samples by discontinuous density gradient ultracentrifugation as previously described.

The LDL was washed at d¼1.063 g/ml, dialyzed against 150 mmol/l NaCI, 1 mmol/l Na2 EDTA (pH 7.4) at 41C.

The LDL fractions were then sterilized by filtration (0.45 mm), kept under nitrogen in the dark at 41C and used within 1 week. The lipoprotein protein concentration was determined by the Lowry assay.22 Prior to oxidation, LDL was dialyzed against EDTA-free, phosphate buffered saline (PBS) solution at pH 7.4, and at 41C. LDL oxidation LDL (100 mg of protein/ml) was incubated with 5 mmol/l of CuSO4 for 2 h at 371C.

Formation of conjugated dienes was continuously monitored by measuring the increase in absorbance at 234 nm.23 Lag time required for initiation of lipoprotein oxidation was calculated from the oxidation curve. The amount of LDL-associated lipid peroxides was measured by the method of El-Saadani et al.20 LDLs isolated from healthy volunteers at the same time periods were used for standardization of the oxidation studies.

Carotid Lesion analyses

Complete atherosclerotic plaques, (including the common, internal and external, carotid parts of the lesion) were collected from 2 groups of patients after endartherectomy. One group included seven patients with CAS that did not consume PJ. The second group included two patients that consumed PJ (for 3 or for 12 months).

The two groups were age matched and had similar serum lipids and glucose concentration. Treatment with PJ was the only relevant difference between these two groups. Lesions were washed in saline, dried, and their weight measured. The lesions were cut into small pieces and rinsed in PBS, followed by their sonication in an ultrasonic processor (320 s at 80 W).

The lesion’s cholesterol content was measured in the homogenate samples by enzymatic, colorimetric assay using commercial kit (Sigma Co. Ltd). The lipid peroxide content in the lesion was also measured.20 Reduced glutathione (GSH) content was measured by the DTNB-GSSG reductase recycling assay.


The ANOVA test was performed for all statistical analyses used to compare repeated measurements. Results are given as mean7SEM. Assays in each sample were performed in triplicate. All comparisons are shown for data after PJ consumption vs. results obtained before treatment


Mean intima-media thickness (IMT) of the left and right common carotid arteries from severe carotid artery stenosis (CAS) patients that did not consume pomegranate juice (PJ), increased significantly (Po0.01), by 9%, during 1 year period from 1.5270.03 to 1.6570.04 mm. In contrast, mean IMT (of the left and right common carotid arteries) in CAS patients that consumed PJ for up to 1 year was reduced after 3, 6, 9 and 12 months of PJ consumption by 13%, 22%, 26% and 35%, respectively, in comparison to baseline values

The inhibitory effect of PJ consumption on carotid PSV was significant only after 1 year, with a reduction in mean PSV of both left and right carotid arteries by 21% (Fig. 1B).

Mean carotid EDV of both left and right carotid arteries however, gradually decreased, by 16%, 20%, 31% and 44%, after 3, 6, 9 and 12 months of PJ consumption, respectively (Fig. 1C).

PJ consumption by the patients did not significantly affect the levels of all major serum biochemical markers studied including: glucose and cholesterol in HDL and LDL (Table 1).

Serum triglyceride concentration however increased by 16%, as reflected by a similar increase in VLDL cholesterol concentration after 12 months of PJ consumption (Table 1), but these increased levels were still in the normal range. Serum markers for heart, kidney and liver function, as well as homocysteine, Lp (a), and total protein concentrations remained unchanged during the whole study

Similarly, blood coagulation and blood cell count were not significantly affected by PJ consumption (data not shown).

The patient’s systolic blood pressure was significantly (Po0.05) reduced by 7%, 11% ,10%, 10% and 12% after 1, 3, 6, 9, and 12 months of PJ consumption, respectively, compared to values obtained before treatment (Table 1).

In contrast, PJ consumption had no significant effect on the patient’s diastolic blood pressure (Table 1). In the control group, systolic and diastolic blood pressure values were not significantly changed along 1 year of follow-up (16077/8874 vs. 16379/8576mmHg at baseline and after 1 year, respectively).

In order to analyze the effect of PJ consumption on the patients’ serum oxidative state, we measured serum concentration of antibodies against oxidized LDL (Ox-LDL).

A significant (Po0.01) reduction in the concentration of antibodies against Ox-LDL by 24% and 19% was observed after 1 and 3 months of PJ consumption, respectively, (from 2070761 EU/ml before treatment to 1563769 and 1670752 EU/ml after 1 and 3 months of PJ consumption, respectively, n¼10). Similarly, total antioxidant status (TAS) in serum was substantially increased, by 130%, from 0.957 0.12 nmol/l at baseline to 2.2070.25nmol/l after 12 months of PJ consumption (n¼10).

These results indicate that PJ administration to the patients substantially reduced their serum oxidative status, and could thus inhibit serum lipid peroxidation. Indeed, serum lipid peroxidation, induced by the free radical generator AAPH, was significantly reduced, by 59%, after 1 year of PJ consumption (from 1670766 to 691743nmol of lipid peroxides/ml, n¼10).

The increased resistance of the patients’ serum to oxidation after PJ administration could have also resulted from increased serum paraoxonase1 (PON1) activity. Fig. 2A demonstrates a significant (Po0.01) increase in serum paraoxonase, measured as arylesterase activity, by up to 83%, after 1 year of PJ consumption (n¼10)

Tuesday, May 24, 2005

Everyday groceries contain ingredients that cause heart disease, diabetes, cancer, osteoporosis and other chronic diseases If you go to your favorite corner drugstore, you’ll find two types of things for sale: 1) processed foods and beverages that cause disease, and 2) prescription drugs that treat the symptoms of those diseases. It’s a brilliant racket: buy the stuff at the front of the store and get diseased, then you become a customer for the drugs sold at the back of the store. That’s called “customer retention” in marketing-speak.

The floor plan of these drug stores even encourages this codependent cycle: the pharmacy is hidden away in the back of the store, forcing customers to walk through aisles loaded with high-impulse junk food items like soft drinks, chocolate bars and snack chips. This is no coincidence: store designers know exactly how to boost impulse sales by forcing customers to navigate through shelves that are intentionally stocked with the most high-profit (and low-nutrition) items available.

And, just to make sure nobody gets away without being at least partly diseased, these drug stores also sell products that claim to be helpful for people with diseases, but that actually give them even more disease. For example, did you ever look at the ingredients on those meal replacement shakes for diabetics? The first three ingredients are water, sugar and sugar (sucrose)! It’s primarily sugar water, sold as a product for diabetics. With products like this on the shelves, even people who think they’re enhancing their nutrition actually end up with a lifelong diabetes problem. (Sugar for diabetics… gee, can it get any crazier?)

On top of all this, virtually every grocery store in America sells a vast array of foods that promote chronic diseases. Foods made with white flour are very common. That includes most breads, cookies, crackers, pastries and pastas. And this one ingredient — refined white flour — strips the body of essential nutrition, leaving it deficient in B vitamins as well as minerals like magnesium and zinc. These are precisely the minerals that children need in order to build healthy nervous systems and avoid behavioral disorders later in life. These are also the same nutrients that pregnant women need to be able to give birth to babies that don’t have birth defects.

Every grocery store in America sells foods containing cancer-causing chemicals (sodium nitrite), heart disease promoting ingredients (hydrogenated oils), and drinks that promote osteoporosis and bone loss (carbonated soft drinks). It’s almost like a disease store, not a grocery store, since most items on the shelves are actually “disease in a box” rather than real food.

In Washington these days, there’s a lot of talk about “protecting Americans.” But I say that if we really want to protect Americans, we need to start banning the food ingredients that are killing Americans. We could save hundreds of thousands of lives each year right here in America if we just banned hydrogenated oils, high-fructose corn syrup, and the refining of whole-grain flour into unhealthful white, bleached flour.

And if we told Americans the truth about their foods — if we gave them honest nutritional advice and dietary advice — we would help people make better decisions that would eliminate chronic disease in their own life, and, for expectant mothers, allow them to give birth to children without birth defects. We need honest food labeling and we need to outlaw toxic ingredients like aspartame, monosodium glutamate, food additives and chemical preservatives like sodium nitrite that directly cause cancers of the digestive tract.

These ingredients are, technically, chemical assaults on the American public. While the Bush administration is out there worrying about biological agents like anthrax and smallpox, people are consuming bacon every morning all across America made with sodium nitrite, a chemical additive that causes colon cancer.

  • And yet the USDA remains silent.
  • The FDA remains silent.
  • The Bush administration remains silent.

And the food companies, playing a full-press game of food politics, continue to insist that all of these ingredients are perfectly good for you, even while suppressing precisely the information that would educate you about ways to enhance your health by making healthier food choices.

Thankfully, there is a growing list of pioneering doctors and researchers out there who are telling the truth about foods; people like Dr. Russell Blaylock, Dr. Gary Null, Dr. Julian Whittaker and Dr. David Williams. Book authors like Dr. Elson Haas and Eric Schlosser.

I feel honored to be joining those people in telling the truth about the dangers of the American food supply and the real cause of chronic disease in this country. Because we have all our priorities mixed up in this country. We’re spending $300+ billion fighting a war in Iraq, and we won’t spend even $100 million a year educating our own people on how they can prevent cancer, diabetes, heart disease, birth defects, osteoporosis, clinical depression, attention deficit hyperactivity disorder and other diseases.

And the decision makers in our federal regulatory agencies who claim to be acting on your behalf are actually censoring the information, making sure the American public doesn’t learn the truth, because the truth would hurt the corporate profits of soft drink companies, food processors, fast food restaurant chains and drug companies. After all, there are lobbyists to please. The public be damned.

To turn the tables on the food lobbyists, and in an effort to educate the public about the details of which grocery ingredients to avoid, I’ve written a book on the subject called “Grocery Warning.” This book reveals all the dangerous ingredients to be avoided, along with which foods and beverages contain them. It presents shopping lists: foods to avoid vs. foods to buy, and helps shopper make informed, health-enhancing foods choices each time they shop for groceries (see related ebook on groceries). You’ll find details on this book at TruthPublishing.com


* Everyday groceries contain ingredients that cause heart disease, diabetes, cancer, osteoporosis and other chronic diseases

Source: http://www.newstarget.com/006760.html


Chelation Therapy

6 May

The Thinking Person’s Guide to Perfect Health

Chelation (pronounced key-lay-shun) is a chemical reaction that results in a bond being formed between a metal ion and an organic (i.e., carbon-based made mostly of carbon) molecule. The resulting complex, metal bound to molecule, is called a “chelate” and contains one or more rings of atoms in which the metal ion is so firmly bound it cannot escape. This allows the metal ion to be transported in the same manner as a prisoner, first handcuffed, then moved from one location to another.

In the presence of aging and disease, the cells’ ability to move metal ions through the system and eliminate them when they are in excess becomes progressively impaired. This is especially true for calcium.

Calcium has vital functions in the human body. Without calcium, teeth and bones could not exist. Nevertheless, as the body ages, lipid peroxidation damages the walls of the arterial tree which is repaired leaving a scar. Then calcium and oxidized cholesterol are incorporated into the resulting scar tissue.

There are several known, and easily avoided, risk factors at work in the creation of arteriosclerosis. Lipid peroxidation begins the inflammatory process in the wall of the artery and is facilitated by the presence of:

  • (1) polyunsaturated fatty acids (present in many “junk-foods”)
  • (2) oxidized cholesterol (from cooked, i.e., pasteurized, milk and other animal foods cooked in open air),
  • (3) the relative absence of antioxidants, such as vitamins A, C and E, and
  • (4) high levels of homocysteine (a condition easily prevented with vitamins B6, B12 and folate). Tobacco smoke drains the body’s resources of antioxidants, particularly vitamin C, and further accelerates arteriosclerosis.

If you know and apply these facts from an early age, there is no reason for arteriosclerosis to develop in your body. “Hardening of the arteries” is not an inevitable disease of aging, as you have been led to believe; it is a disease of bad habits. To know and apply these facts, you have to be willing to think for yourself and ah, there is the reason arteriosclerosis will continue to kill people. Maybe the good do die young but so do the uninformed and dogmatic.

As the years pass, calcium deposits build up, and in association with cholesterol, calcified plaques and atherosclerotic plaques form, lining the arterial vessels. When calcium predominates, this process is called “hardening of the arteries” or arteriosclerosis, and when cholesterol predominates it is called “atherosclerosis.” The exact content of the plaques is determined by the individual’s diet, antioxidant intake and duration of the process. Regardless of where on the arterio-/athero- sclerotic continuum any particular individual falls, the result is the same: less and less fresh oxygen delivered to the tissues of the body.

It once was thought this process began in middle or old age. It is now known to begin in childhood in many people. The severity of this life-long process is determined by genetics, level of exercise and dietary habits. By age 21, many individuals have arterial disease, easily recognized at surgery or autopsy.

This is a disease of modern civilization. Never before have people so young had arteriosclerosis. As recently as the year 1900, heart disease was very rare. It may be that airborne industrial pollutants, as well as herbicides, pesticides and preservatives in our food, have a lot to do with arterio-/athero-sclerosis.

The cholesterol content of these plaques can be handled by shifting to a no-fat, high-fiber diet. Plaques actually decrease in size, and the cholesterol content can eventually disappear. Lipid peroxidation itself can be halted by the liberal intake of antioxidants such as Beta-carotene (the precursor of vitamin A), mixed tocopherols (vitamin E) and vitamin C, so no further damage is caused to the arterial tree.

The calcium content of the scar-plaques already present is another matter. Diet and pure water have little effect on it. Therefore, if you want to restore your health to a completely youthful condition, you are facing a real challenge with arteriosclerosis.

The list of problems that can be caused by artery disease is truly impressive, but it should not be surprising that it is so extensive given that a fresh supply of oxygenated blood is absolutely necessary for proper functioning in any organ. Even diseases that are more complicated, in that they have causes other than decreased blood flow, are made worse by arterial disease.

A prime example is Alzheimer’s Disease. True Alzheimer’s Disease is mimicked by simple aterio-/athero-sclerosis of the arteries and arterioles supplying the brain. Diabetes is known to be made worse by poor blood flow to the pancreas, and poor blood supply also can cause decreased output of digestive enzymes from the exocrine part of the pancreas, causing incomplete digestion.

Decreased blood supply to the kidneys results in the inappropriate release of angiotensin by the kidneys, inducing hypertension throughout the vascular tree. The joints, particularly the joints of the low back, react with inflammation and pain to decreasing blood flow and this, along with the degeneration of ligament tissue and disc disease, is responsible for the so-called “low back syndrome.”

Arterio-/athero-sclerosis plays a big part in the cause of arthritis throughout the body. The effect of this process on the heart is angina (chest pain originating in the heart) and eventually infarction and death. Poor blood supply to the stomach and small intestines results in poor digestion. Poor blood supply to the colon causes slowing of the colon with resulting colon disease.

The effect on the extremities is cold hands and feet, and in an advanced case, gangrene of the extremities can result. Impotence can be caused by decreased blood flow to the penis due to clogged arterioles. Frigidity can be caused by decreased blood flow to the pelvis. Cancer is known to be accelerated by decreased blood flow to the affected tissues. When blood flow is decreased to the immunocompetent cells in the bone marrow and spleen, the immune system itself is weakened.

The list of pains, aches, discomforts and diseases caused, or made worse by, arterio-/athero-sclerosis goes on and on. The above discussion is not complete and could not be made complete unless expanded to book size. Fortunately, there is a way to deal with arteriosclerosis. The answer is chelation.

Prevention of Arteriosclerosis: Oral Chelation

An ounce of prevention certainly is worth a pound of cure. The oral chelating agents serve admirably to prevent or halt the progression of arteriosclerosis, but do little to reverse the disease once it is present. You probably already are taking one of the oral chelating agents, vitamin C. This is an excellent oral chelating agent and also easily available. Also, fresh vegetables are loaded with other natural and effective chelating agents.

Exercise-generated Chelation

Lactic acid, produced from exercise, is an excellent chelating agent. It is the metabolic byproduct of sustained, vigorous muscle contraction. To get this chelating agent, you must exercise regularly. Exercise also increases your body’s ability to reduce, and thus neutralize, free radicals, which are at the heart of degenerative diseases.

There is a host of more exotic substances (Anginin, Unithol, Vaso Elastin, DMS, NTA, Hexopal Forte, Syntrival) that I think you should ignore, since they are not readily available, they are expensive, and the agents already easily available to you are excellent.

Reversal of the Effects of Arteriosclerosis by Intravenous Chelation

In distinction to the oral chelating agents that serve to prevent arteriosclerosis, intravenous chelation has been shown to actually reverse the effects of the disease. The agent used is Ethylene-diamine-tetra-acetic Acid, also known as “EDTA,” sold commercially as Sodium Edetate.

EDTA is a synthetic amino acid. The usual dose is 2000-3000 mg. (adjusted to body weight, age, and kidney function) added to 500 ml. of “carrier solution” sterile water with a mixture of vitamins and minerals. Most chelation doctors add vitamin C along with B vitamins, bicarbonate and magnesium.

The solution is infused slowly, one drop per second, and one treatment requires about three hours. The prisoner (calcium) is moved out of the body using the sheriff’s handcuffs (EDTA). The half life of EDTA in the body is one hour; i.e., one-half is removed (filtered into the urine) after one hour, another half of what is left is removed after one more hour, etc. Within 24 hours 99% of the EDTA is gone from the body, and you are left with only the therapeutic benefit.

In addition, to the transitory transport of calcium, many other metal ions are transported and rearranged, which brings up the subject of how EDTA works. In the early days of EDTA therapy, physicians had no idea how it worked. As physicians do, they reached for the nearest reasonable explanation. They said it decalcified the walls of arteries clogged with arterio-/athero-sclerotic plaque, a kind of chemical ROTO-ROOTER ®. This is now known not to be the only benefit of EDTA, even though decalcification of plaques does occur. The action of EDTA is more complex than the simple-minded comparison with a ROTO-ROOTER can reflect.

To be sure, the action of EDTA is to increase blood flow throughout the body. One of the hallmarks of aging is decreased blood flow to all the organs. It has been shown conclusively: EDTA restores this lost blood flow. How can this happen, if EDTA is not a “ROTO-ROOTER?”

Delivery of oxygen to cells is not explainable by merely comparing the circulatory system to a set of pipes. Blood vessels are living organs, not pipes. Once oxygen is delivered to a cell there is still the matter of how efficiently it can be used. EDTA, as it turns out, operates at all these levels. Here are the effects of EDTA, the final manifestation of which is the healing of degenerative diseases of many kinds.

  • 1. EDTA lowers blood calcium and thus stimulates the production of parathormone from the parathyroid glands. This mild pulse of parathormone is responsible for the removal of calcium from abnormal locations (such as arteries) and the deposition of calcium in locations (such as bones) where it should be. This accounts for the mild recalcification of osteoporotic bones seen with EDTA.
  • 2. EDTA stimulates the enlargement of small vessels, so that they serve the purpose of collateral circulation around a blockage, rendering the blockage irrelevant.
  • 3. EDTA controls free radical damage due to lipid peroxidation by serving as a powerful antioxidant.
  • 4. EDTA removes abnormally located metal ions, such as copper and iron, that accumulate with age.
  • 5. EDTA removes lead, cadmium, aluminum, mercury and other metals, restoring enzyme systems to their proper functions.
  • 6. EDTA enhances the integrity of cellular and mitochondrial membranes.
  • 7. EDTA helps reestablish prostaglandin hormone balance. Prostaglandins, among other things, are responsible for the balancing act between contraction and relaxation of arterial walls and between clotting and the free flow of blood. Prostaglandins are produced from fatty acids, therefore lipid peroxidation upsets the balance of these vital hormones. EDTA chelates out the catalyzing metallic co-enzymes and thus inhibits lipid peroxidation, also serving the same function as an antioxidant.
  • 8. EDTA reduces the tendency of platelets to cause coagulation too readily. This tends to prevent inappropriate thrombosis, which blocks coronary arteries during a heart attack.
  • 9. EDTA increases tissue flexibility by uncoupling age-related cross-linkages that are responsible for loss of skin tone and for wrinkling.

I recommend any individual over the age of forty to have a series of twenty EDTA treatments, followed by six to twelve per year for maintenance after that, simply to restore youthful vitality lost due to aging and arteriosclerosis. A person who is already symptomatic with a cardiovascular disease will require more than twenty treatments. We look for the end of troublesome symptoms such as chest pain, leg pain, transient dizziness, intellectual impairment, and fatigue all attributable to loss of blood flow to vital organs to know when there have been enough treatments. A good rule of thumb to estimate the maximum number of treatments needed is one treatment for every year of your age, minus 20, but this is only a rough estimate.

You should expect to pay $90-120 per treatment, which admittedly is a nice piece of change. Most people would spend more money on a new car than on their health, so you have to ask yourself how much your health is worth. In the long run, the money you spend on chelation should more than repay itself in health, vitality and the absence of illness. If this were not so, I would not recommend it to you, and I would not be a chelation therapist. The number of physicians who carry out this procedure is relatively small, but growing rapidly a few hundred in the U.S. at present. This relative unavailability is surprising, given the great benefits available through this relatively inexpensive, extremely safe treatment.

A Short History of EDTA

EDTA was developed in Germany in the early 1930s as a substitute for citric acid. Citric acid was produced in England and used by Germany for binding mordant dyes. The development of EDTA was part of Germany’s effort to become independent of other countries. No one dreamed at the time that it would ever have a medical use. It has been available in the U.S. for medical purposes since 1948. The controversy has been raging since then, and it is not going away, much to the chagrin of the medical/pharmaceutical complex.

Background Information

Many physicians who administer EDTA are people who have benefited from it themselves, many of whom have been brought back from death’s door, most commonly from heart disease. As I write this, I am experiencing the absence of a severe low back pain condition, which had been with me for thirteen years, relief I attribute to EDTA! Also my hearing, which was beginning to fail, has cleared up dramatically, and my kids are now puzzled that I can hear them from the other room.

I was introduced to EDTA by an 84-year-old former surgeon, Martin Weiss, M.D., who had been given a death sentence by a cardiovascular surgeon at age 67 unless he would immediately undergo coronary bypass. He knew the dangers of surgery and looked around for an alternative. He learned of EDTA and through treatment became free of heart disease without the risk of anesthesia or surgery. He then decided to offer EDTA to his patients.

Many physicians are closet chelators who perform chelations on themselves and their loved ones and relatives, but do not offer it to the general public because of the threat of condemnation by the medical community. These physicians are severely constrained by their need to be accepted by their peers. The freedoms we enjoy in America were not won by such people.

Medical Politics

One can speculate about why this treatment is not more well-known and commonly administered in modern medicine in the U.S. It is interesting to observe, the patent on EDTA ran out in 1948, and it is therefore very inexpensive, because it can be produced by any drug company and must therefore face free market competition. It hardly matters how effective any drug is, when the patent expires, you probably will not hear much more about it. Drug companies have no fortune to make and thus no motivation to advertise EDTA to doctors. This kind of advertisement, believe it or not, is the most important factor determining which drugs many doctors prescribe, because it is this advertising doctors rely on for the bulk of their “continuing education.”

Also, if EDTA became commonly used, there would be a lot of cardiovascular surgeons looking for something else to do, as EDTA is a reasonably priced (cost: $2,000-4000), safe, nonsurgical alternative to balloon angioplasty (cost: around $15,000), and coronary bypass operations (cost: in the range of $50,000!).

Many of these surgeons make over two million dollars per year doing drastic procedures for illnesses which could have been prevented with oral chelation, and many even most of which can still be treated successfully with EDTA. If these surgeons go out of business so does a section of hospital surgery suites and with those, many hospitals. The economic phalanx lined up against chelation therapy is solid and deep.

It is interesting to note a recent study in a publication called Medical Care (1995;33(7):715-728). This study reports that coronary bypass surgery is 96% more likely to be recommended when the patient is covered by private insurance versus Medicare (which pays less), and 117% more likely to be recommended versus the noninsured (which pays even less).

I recently attended the thirty year reunion at the university where I took my premed training. There I met an old friend who had become a vascular surgeon. This man was a wonderful student who never made less than an “A” on any test. I thought that, of all people, Ed would have looked over the relevant studies and would have a well-thought-out opinion for or against bypass surgery.

So, I asked him, “Ed, what do you think of bypass surgery now? Is that good for people? Should we be doing that to people?” His reply: “It pays the bills!” And that was it. I could not persuade him to say anything more about the matter. He did offer that he was looking forward to an early retirement, but he had no more to say about bypass surgery.

One can only speculate about why the mass consciousness of doctors is not simply neutral to EDTA, but is, instead, openly hostile and disparaging. Otherwise open-minded docs will say absurd things like “I don’t know anything about it except it is no good!” How can you know it is no good, if you know nothing about it? My guess is: it is a combination of unconsciousness, ignorance and pure capitalism on the part of both pharmaceutical companies and medical practitioners.

Many courageous physicians have faced censure from medical societies, loss of hospital privileges, and worse for administration of this incredibly effective and safe treatment. Those days are coming to an end, however, because of the massive evidence which has accumulated to validate the safety and effectiveness of EDTA and the power of ACAM, the medical society for chelating doctors.

Nevertheless, we cannot take this therapy for granted. The California Medical Board is striving, right now, 1996, to regulate the use of EDTA to the point that it will not be available for the conditions for which most people need it. The Board is evenly split on whether to do this or not with (predictably) the vascular surgeon on the Board rabidly for suppression of chelation, despite the evidence of its effectiveness.

As one of these rigid, righteous, closed-minded doctors said at a recent board meeting, “As long as chelation therapy was limited to being used by only a few docs, it did not need to be regulated, but now that it is becoming well-known, this ripoff therapy must be suppressed.” What he did not say, that is clearly true, is he wants to stamp out the competition to his enormous coronary bypass fees. This meeting was open to the public, and the room was full of hundreds of people whose lives had been saved by chelation, one of whom shouted out “Coronary bypass is the real ripoff!”

Let me quote this surgeon a little more. “If EDTA is so good, let them prove it. Proof is not so hard to get! Let them prove it with controlled, double-blind, placebo studies and then publish these results in the top peer-reviewed journals.” He apparently had an attack of attention deficit disorder when these very studies had been presented to the Board only a few minutes before.

Only a few of the thousands of fine studies on EDTA have been published in what were once the distinguished journals of medical research. The reason for this: the pharmaceutical industry bought these journals out with “donations” and advertising dollars years ago. Studies on the uses of EDTA threaten the profits of the pharmaceutical industry with its panoply of patented, toxic, synthetic drugs and the surgical industry with its dangerous unnecessary interventions such as bypass surgery.

These studies simply are not allowed to be published in what were once the best medical journals, but that now are disrespected by doctors who are knowledgeable about the political process behind these publications.

Indeed, the surgical, pharmaceutical and hospital industries would like to stamp out chelation therapy. I am sure some people at MacDonald’s would like to outlaw other restaurants and make the Big Mac the required “food” for every person on the planet. Quality meals, like quality medical care, are not served at every standardized outlet.

A Vignette

Ten days ago one of my patients finished his course of chelation therapy. He went back for a visit to his cardiologist, who had recommended angioplasty and who strongly opposes chelation therapy. This man informed my patient that chelation therapy is dangerous, unproven, a financial ripoff and then insisted that my patient get back on his Mevacor (a toxic synthetic drug for lowering cholesterol).

He then mailed to me a nasty little “progress note.” A few days later my patient dropped by my office for a chat and pointed out that as a result of chelation therapy his blood pressure is down, his diabetes is under control, his arrythmia is no longer present, and he has a new-found experience of well-being.

When informed by his cardiologist that my fee was a ripoff, my patient reports that he leaned toward that doctor and asked “Just how much does angioplasty cost?” (Answer: $20,000 for a two-hour procedure which typically is a failure versus my fee of less than $2,300 for 25 three-hour chelation treatments which typically are successful.) Almost any chelation therapist can tell you several such stories.

I am proud to be a physician. I studied and worked hard for my degree. The only time I am embarrassed to be a doctor is when I see performances like this one by a colleague. I am embarrassed that this man has the same degree I have. I know better than to hope this doctor will change. The facts do not matter to righteous, closed minds. Things will change, but as a result of people like that growing rich, old, retired, and replaced by a new generation.

Insurance Politics

Insurance companies, including Medicare, will not cover the cost of chelation therapy with EDTA, even though the cost is only around $3,000 compared to $15,000 for angioplasty and $50,000 for a bypass. The excuse is, EDTA is not an “accepted” therapy.

What that actually means is: not accepted by cardiovascular surgeons who compete with chelation therapy and not accepted by the drug industry, which depends on people remaining sick and taking loads of synthetic drugs, and not accepted by the leading medical journals, which have been bought out by the pharmaceutical and surgical industries.

What is most strange, on the surface, is the fact that insurance companies do not cover the costs of chelation, even though they will shell out for coronary bypass which costs fifteen times as much and treats only two, three or four of the hundreds of arteries in the body.

However, if you consider how widespread is the incidence of arterio-/athero-sclerosis, the number of insured people who would need EDTA as a preventive measure is truly astounding, and the cost of covering those people is clearly outside what is possible for any insurance carrier. Perhaps Medicare and the insurance companies have thought rather deeply into what it would cost to cover chelation therapy.

Nevertheless, if you are willing to have your treatment and then sue your insurance company for coverage, you probably will win, provided you present the facts about EDTA clearly. Historically this has been the case. I have a stack of several hundred scientific articles on EDTA, and I am prepared to prove my point in any forum.


* Clarke NE, Clark CN, Mosher RE The “in vivo” dissolution of metastatic calcium: An approach to atherosclerosis. Am J Med Sci 1955;229:142-149.
* Clarke NE, Clark CN, Mosher RE Treatment of angina pectoris with disodium ethylene diamine tetraacetic acid. Am J Med Sci 1956;232:654-666.
* Lamar CP Chelation therapy of occulsive artherosclerosis. J Am Geriatr Soc 1966;14:272-293.
* Bjorksten J The cross-linkage theory of aging as a predictive indicator. Rejuvenation 1980:8:59-66.
* Blumer W, Reich T Leaded gasoline – a cause of cancer. Environmental International, 1980;3:456 -471.
* Casdorph HR, Farr CH EDTA chelation therapy III: Treatment of peripheral arterial occlusion, an alternative to amputation. J Holistic Med 1983;5(1):3-15
* Selhub J, et al. Association between plasma homocysteine concentrations and extracranial carotid artery stenosis. N Eng J o Med 1995; 332:286-291

Source Page: http://www.contextpub.com/ex_tpg_chelatio.html

Wheatgrass – Amazing Graze

4 May

Wheatgrass - Amazing Graze Gifts

Amazing Graze Gifts

There’s nothing like a shot of wheatgrass juice to lift your spirits. Not only does it help treat disorders or diseases of the body, but it extends also to the emotional and mental aspects of the individual.
But what is it about this drink that brightens up your mood?

The magic ingredients are the many bio-active substances produced when wheat is sprouted (germinated), and allowed to grow to the height of 6 inches or more, usually for 7-14 days (depending on warmth). Wheatgrass, grown in highly mineralized soil can pick up nutrients from the soil and store them in the juices of the young leaves.
The secret of its amazing healing powers is situated in the leaf – the specific sun-trap is the plant pigment chlorophyll which traps the energy of sunlight for photosynthesis. It is a green substance which contains molecules of the silvery-white metal magnesium. When photons of light are absorbed by the blades, a radiative excitation occurs resulting in the many dynamic changes which take place in the early life of the wheat plant. Chlorophyll constitutes 70% of the solute in wheatgrass and has been extensively tested for its extraordinary healing powers. The following are a few examples of what happens during this time:

* A marked increase in vitamin and mineral production. Anti-oxidant activity in wheat sprouts and young grass as well; these include traces of beta-carotene (pro vitamin A), vitamins C and E. Vitamin C value in wheatgrass increases 600% during the first few days of germination. Vitamin E content by 300%.
* High enzyme content. Enzymes are fragile and break down at temperatures in excess of approximately 106 – 130 degrees F, so we won’t find them in cooked food.

* Dr. E. Pfeiffer, a biodynamic agricultural researcher, proved that on the 8th day of growth, all essential and 17 total amino acids are present in wheatgrass.
* Alkaline minerals, calcium, potassium and magnesium(among others) are also produced during this time which helps to ensure that wheatgrass is an alkaline forming food.

Magnesium, which is the central atom of the chlorophyll molecule, aids calcium and other elements to be absorbed by the bones. Industrial magnesium burns with an intense white flame, and is used in flash bulbs, flares and fireworks.
Soil should be tested when food is grown on a large scale, as magnesium levels do not remain constant. As a result, many store-bought greens fail to reflect adequate magnesium content.

According to European studies 87% of the population has an inadequate magnesium intake.
Depleted magnesium reserves in the body can lead to a variety of illnesses, including cramps, heart problems and an overtaxed nervous system. Psychological changes such as apathy, lack of concentration, anxiety, insomnia and depression may also result.

You do not have to embark on a stringent dietary regime to benefit from wheatgrass. Just one glass of wheatgrass juice a day works wonders!

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Wheatgrass: Superfood for a New Millennium

4 May

Li D SmithBeing alert and alive turns you into a happy walking, talking ball of energy. You will also feel much better. Complete rejuvenation of body, mind and spirit is possible when vitality is high.

Scientists agree: you are your best healer. Studies show that you can overcome chronic health problems including high blood pressure, asthma, depression, certain types of cancer, diabetes and osteoporosis with good nutrition and sensible exercise.

All living things need energy to live. Both animals and plants need a supply of raw, live food to provide them with this energy. Radiant energy of sunlight and chemical energy of organic compounds are the two sources of energy available to living systems. Green plants, algae and some bacteria are able to convert this energy, through photosynthesis, into chemical energy that they can use. Stored as simple sugars, it provides the necessary energy for important metabolic activities essential for growth, repair and maintenance. Energy is created in every cell, everyday. Each cell requires nutrients, with some being used as “fuel” for cellular respiration.

Wheatgrass is a natural energizer that really works. In wheatgrass, photosynthesis takes place in the chloroplasts, miniature solar converters inside the plant cells. The chloroplasts contain the pigment chlorophyll, which is able to absorb light in the blue, violet and red parts of the spectrum. Green light is not absorbed, but reflected. This is why wheatgrass appears green.

If you are not as fit and healthy as you’d like to be, consider drinking wheatgrass juice on a daily basis. Wheatgrass juice is the all-in-one solution to radiant health, energy and rejuvenation!

Wheatgrass, which is the young grass of the wheat plant, captures an enormous amount of radiant energy via photosynthesis. Chlorophyll constitutes 70% of the solute in wheatgrass and has been extensively tested for its extraordinary healing powers.

Generous amounts of vitamins are also present in fresh, healthy wheatgrass. The antioxidants, A, C and E are abundant, as are minerals of key importance such as magnesium, calcium, iron, zinc and potassium. A glass of the green-juice supplies you with almost all of the 102 known elements (that is, if it is grown in optimal organic soil). It is a complete food!

Author Li D Smith of “Wheatgrass: Superfood for a New Millennium” was born in Luxembourg and raised on a farm in South Africa, married an artist and enjoys painting and writing.

Great health doesn’t require bags full of expensive supplements. An abundance of positive energy, and a glass of wheatgrass juice is often all you need to set the scene for a healthier life.

The following are links to Li D Smith and Richard’s bio and book pages,
A MUST BUY For D-Light-full Health!

Li D Smith with Wheatgrass Tonic

Author Li D Smith
Wheatgrass: Superfood for a New Millennium

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